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1、·606·中国呼吸与危重监护杂志2009年11月第8卷第6期ChinJRespirCritCareMed,November2009,Vol18,No16orTalleleatthesingle2nucleotidepolymorphismatposition1580:2008,5:3482353.implicationsfordisease.BiochemJ,2003,369:1792184.22WangX,DalkicE,WuM,etal.Genemodulelevelanalysis:16WurfelMM,GordonAC,HoldenTD,etal.Toll2likerecept
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5、DN,etal.MicroarrayanalysisofLungCellMolPhysiol,2007,293:L2922302.regionalcellularresponsestolocalmechanicalstressinacutelung20dosSantosCC,OkutaniD,HuP,etal.Differentialgeneprofilingininjury.AmJPhysiolLungCellMolPhysiol,2006,291:L8512861.acutelunginjuryidentifiesinjury2specificgeneexpression.Crit
6、27KampR,SunX,GarciaJG.Makinggenomicsfunctional:CareMed,2008,36:8552865.decipheringthegeneticsofacutelunginjury.ProcAmThoracSoc,21KampR,SunX,GarciaJG.Makinggenomicsfunctional:2008,5:3482353.decipheringthegeneticsofacutelunginjury.ProcAmThoracSoc,(收稿日期:2009202224)·综述·慢性阻塞性肺疾病相关性贫血陈红邓佳冯玉麟四川大学华西医院(四
7、川成都610041)COPD是一种可预防、可治疗,以气流不完全可逆受限长期家庭氧疗(LTOT)的重度COPD患者,贫血患病率为:男[6]并呈进行性发展为特征的疾病,与肺部对有害气体或有毒颗性1216%,女性812%。John等回顾性地调查了1996～粒的异常炎症反应有关。在全球范围内COPD是引起死亡2003年德国柏林各大学慈善医院因COPD住院的312例患[7]和功能致残的主要疾病之一。COPD在全球患病率和死亡者,贫血患病率为2311%。S
