肝脏交感神经调控与急性肝损伤关系的实验研究

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3、联免疫吸附测定氯化六烃季铵小时免疫组织化学白细胞介素．1B枯否细胞脂多糖丝裂原蛋白激酶分钟毫升多系统脏器功能衰竭信使RNA去甲肾上腺素核因子．KB纳克光密度磷酸盐缓冲液哪8㈣9Ⅲjl㈣2㈣0舢8iiii●IⅢY第二军医人学硕士论文pgPARNAPCRSecsystemicTBTLR4PicogramprealbuminRibonucleicacidPolymerasechainreactionSecondinflammatoryresponsesyndrome，SIRStotalbilirubinToll··likereceptor--4TNF—QTumornecrosisf

4、actor-QTPtotalTransectionUlprotein皮克前白蛋白核糖核酸多聚酶链反应秒全身炎症反应综合征总胆红素Toll样受体4肿瘤坏死因子．Q总蛋白ofcervicalsympathetictrunkgroups，TCST颈交感神经干离断Microlitre2微升第三军医人学硕士论文ThestudyofrelationshipbetweenhepaticsympatheticBackground：-●·_●●regulationandacute1iverln．1uryAbstractIntheclinic，acuteliverinjuryduetOserio

5、ustrauma，majorsurgery,poisoning，bacteria，virusinfectionmayeasilyleadtOacuteliverfailure．Atpresent，thedrugtreatmentishardtOgetanidealeffect．Inthispathologicalprocess，duetotrauma，bacteria，viruses，etcasadirectresultoflivercelldamageislimited．Infact，thebody’Sinappropriateinflammatoryorimmuneres

6、ponsetOpathogensmayresultinseriousconsequences．Excessiveinflammatoryresponsecausedbyalargenumberofinflammatorymediatorsinthebloodcirculationmayendinsystemicinflammatoryresponsesyndrome(SIRS)evenMOSEacuteliverinjuryisonepartofit．TheoccurrenceanddevelopmentofInflammationisacomplexprocesswhich

7、affectedbymanyfactorsregulation．Inflammatoryregulationcanbebroadlydividedintotwomajorcategories：bodyfluidsregulationandneuralregulation．Atpresent，theresearchofinflammatoryresponseandliverinjuryfocusedonfluidconditioning，thedataofneuralregulationshowlessf