欢迎来到天天文库
浏览记录
ID:20544033
大小:53.00 KB
页数:7页
时间:2018-10-13
《大黄牡蛎汤灌肠治疗痛风性肾病的临床观察》由会员上传分享,免费在线阅读,更多相关内容在工程资料-天天文库。
1、大黄牡蛎汤灌肠治疗痛风性肾病的临床观察摘要:目的研究大黄牡蛎汤灌肠治疗痛风性肾病的临床效果,为临床痛风性肾病的治疗提供依据。方法将86例痛风性肾病患者随机分为2组,治疗组46例,对照组40例,2组患者均予低盐低嘌呤饮食,对照组予别嘌呤醇、碳酸氢钠片口服,治疗组则在对照组基础上加用大黄牡蛎汤保留灌肠,观察2组治疗前后患者血尿酸、血肌酐、尿素氮及24h尿蛋白定量。结果治疗组总有效率为8913%,对照组总有效率为725%,2组比较,右显著性差异(P〈005)。治疗后2组均能明显降低血尿酸水平,组间比较无显著性差异(P>005);但
2、在改善肾功能及尿蛋白水平方面,治疗组疗效优于对照组(P<005)o结论大黄牡蛎汤保留灌肠治疗痛风性肾病,对于降低血尿酸及减轻蛋G尿,延缓肾功能损害等方面疗效显著,无明显不良反应,值得临床推广。本文采集自网络,本站发布的论文均是优质论文,供学习和研究使用,文中立场与本网站无关,版权和著作权归原作者所有,如有不愿意被转载的情况,请通知我们删除己转载的信息,如果需要分享,请保留本段说明。关键词:大黄牡蛎汤;灌肠;痛风性肾病;肾功能;血尿酸;蛋白尿中图分类号:R692文献标志码:B文章编号:1007-2349(2017)07-004
3、5-03LAbstract]Objective:TostudytheclinicaleffectofRhubarbOysterDecoctiononthetreatmentofgoutynephropathy,andtoprovidethebasisforthetreatmentofclinicalgoutynephropathyMethods:86patientswithgoutynephropathywererandomlydividedintoatreatmentgroup(n=46)andacontrolgroup(
4、n=40)AllthepatientsweregivenlowsaltandhypopurinedietThecontrolgroupwastreatedwithallopurinolandsodiumbicarbonatetabletswhilethetreatmentgroupwasgivenretentionenematreatmentwithrhubarboystersouponthebasisoftreatmentofthecontrolgroupThelevelsofserumuricacid,serumcrea
5、tinine,urinenitrogenand24-hoururineproteinwereobservedbeforeandaftertreatmentResults:Thetotaleffectiverateofthetreatmentgroupwas8913%whilethatofthecontrolgroupwas725%Therewassignificantdifferencebetweenthetwogroups(P<005)Thelevelsofserumuricacidofthetwogroupsdecrea
6、sedaftertreatment,buttherewasnosignificantdifferencebetweenthetwogroups(P<005)However,thecurativeeffectofthetreatmentgroupwasbetterthanthatofthecontrolgroup(P<005)intheimprovementofrenalfunctionandurineprotein,buttherewasnosignificantdifferencebetweenthetwogroups(P
7、>005)Conclusion:RhubarbOysterDecoxtionretentionenematreatmentissignificantlyeffectivetogoutynephropathyandcanreduceblooduricacidandproteinuriaanddelayrenalfunctiondamage,nosignificantadversereactionsandworthyofwideclinicalapplication[Keywords]RhubarbOysterDecoction
8、,enema,goutynephropathy,renalfunction,uricacid,proteinuria痛?L性肾病是由于体内嘌呤代谢紊乱,血尿酸生成过多或排泄减少导致血尿酸升高,过多的尿酸盐在肾脏发生结晶沉积于肾间质、肾小管腔内,引起肾脏组织结构与肾功能损害的病变,迁延可发展
此文档下载收益归作者所有