克瘤丸对晚期非小细胞肺癌生长转移及血管生成的影响

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1、ABSTRACTMTTassayintriplicate．(乳heeffectofKELIUWANonvesselgrowthwasobservedbyadoptingchickembryochorioallantoicmembrane，CAMmodel③ToobservetheimpactofKeLIUWANonthegrowthandmetastasisandexpressionofVEGFandMVD，Lewislungcancermodelwasadopted．@Acutetoxicitytest

2、ing．Results1．Theoreticalresearchresults：OnthebasisofliteratureandtheideologyofmymentorYonghongZHANG；wearguethatvitalQideficiencyisthepathogenesisbasisandstagnationofphlegm，bloodstasis，poisonisthekeyfactoroflungcancer,andthetreatmentprincipleshouldberemovi

3、ngbloodstasistodispelnodes，reinforcingQiandrelievingpoison．2．Clinicalresearchresults：①diseaseremissionrate(CR+PR)，theremissionrateofgroupsBandCwerehigherthangroupA，andthedifferencehasstatisticalsignificance(P<0．01)；②diseasecontrolrate(CR+PR+SD)，diseasecon

4、trolrateofgroupCWashigherthangroupAandB，andthedifferencehasstatisticalsignificance(P

5、mentofclinicalsymptomsandsigns．TheefficiencyrateofgroupAandBwerehigherthangroupB，andthedifferencehasstatisticalsignificance(P<0．01)；⑤improvementrateofsinglesymptom．Theimprovementrateofcough，chestpainandshortofbreathingroupCWasstatisticallydifferentfromgro

6、upB(P<0．05)；⑥improvementofcommonconditions．TheKamofskyscoresstabilityrate(ascendandstable)ingroupAwashigherthangroupB，andthedifferencehasstatisticalsignificance(P<0．05)；⑦theVEGFvaluedifferencesbeforeandaftertreatmentingroupAandBhasstatisticalsignificance(

7、P<0．05)，whilethoseingroupBdonot(P>O．05)．⑧inpatientsachievedPRandSD，VEGFvaluedingroupAandBdeclinedremarkablycompared、)~，ithbeforetreatment，whichhasstatisticalsignificance(P<0．05)，whileingroupBtheVEGFvaluesdifferencesdonothavestatisticalsignificance(P>0．05)

8、．3．experimentalresearchresults：①thecomparisonoftheinhibitoryinfluenceofthethreedistillmethodsofKELIUWANontheproliferationofhumanlungcancercelllineA549：theinhibitoryefficiencyofKELIUWANby’’integratedextractedmethod’’