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时间:2020-06-03
《电刺激延髓外侧网状核对大鼠心脏一躯体运动反射的下行性抑制作用及其机制.pdf》由会员上传分享,免费在线阅读,更多相关内容在行业资料-天天文库。
1、第38卷第3期吉林大学学报(医学版)Vo1.38No.32012年5月JournalofJilinUniversity(MedicineEdition)Mav2012433[文章编号]1671—587X(2012)03—0433—05电刺激延髓外侧网状核对大鼠心脏一躯体运动反射的下行性抑制作用及其机制韩曼,刘晓华,孙娜。,杜剑青(1.陕西中医学院生理学教研室,陕西咸阳712046;2.西安交通大学医学院生理学与病理生理学系,陕西西安710061)[摘要]目的:观察电刺激延髓外侧网状核(LRN)对大鼠心包内辣椒素(IC)诱发心脏一躯体运动反射(CMR)的下行性抑制作用并探讨其
2、作用机制。方法:22只雄性SD大鼠随机分为电刺激组、电刺激+鞘内注射3g·L育亨宾组、电刺激+鞘内注射5g·L育亨宾组和电刺激+鞘内注射溶媒组,观察各组处理因素干预后大鼠Ic诱发CMR的脊斜方肌肌电(EMG)的变化。结果:与对照组比较,电刺激(10、20和30tLA)LRN能够抑制EMG,随着刺激强度的增加,抑制效果也加强,EMG分别下降到对照组的51.41、21.11和1.43,(P3、72.4O和78.73(P4、mentofPhysiology,ShaanxiUniversityofChineseMedicine,Xianyang712046,China;2.DepartmentofPhysiologyandPathophysiology,SchoolofMedicine,Xi’anJiaotongUniversity,Xi’an710061,China)Abstract:ObjectiveTostudythedescendinginhibitoryeffectofthelateralreticularnucleus(LRN)treatedwithelectrica1stimula5、tionontheeardiosomaticmotorreflex(CMR)inducedbyintrapericardia1capsaicin(IC)inratsanditsmechanism.Methods22maleSDratswererandomlydividedintoelectrica1stimulationgroup(一7),electricalstimulationplus3g·Lyohimbinegroup(n一5),electricalstimulationplus5g·Lyohimbinegroup(,2—5)andelectricalstimulat6、ionplusvehiclegroup(一5).Theelectromyogram(EMG)changesofdorsalspinotrapeziusmuscleofCMRinducedbyICofratswereobservedfollowinginterventionwithtreatmentfactorsinvariousgroups.ResultsComparedwithcontrolgroup,EMGcouldbeinhibitedbyelectricalstimulationof10,20and30AinLRN,theinhibitoryeffectwasinc7、reasedwiththeincreasingoftheelectricalstimulationintensityaccompaniedornotaccompaniedwithchangesofbloodpressure;theEMGreducedtO51.41,21.11and1.43ofthecontro1(P<0.001);Comparedwithelectricalstimulation(10“A)group,intrathecalinjectionofadrenergica2receptorantago
3、72.4O和78.73(P4、mentofPhysiology,ShaanxiUniversityofChineseMedicine,Xianyang712046,China;2.DepartmentofPhysiologyandPathophysiology,SchoolofMedicine,Xi’anJiaotongUniversity,Xi’an710061,China)Abstract:ObjectiveTostudythedescendinginhibitoryeffectofthelateralreticularnucleus(LRN)treatedwithelectrica1stimula5、tionontheeardiosomaticmotorreflex(CMR)inducedbyintrapericardia1capsaicin(IC)inratsanditsmechanism.Methods22maleSDratswererandomlydividedintoelectrica1stimulationgroup(一7),electricalstimulationplus3g·Lyohimbinegroup(n一5),electricalstimulationplus5g·Lyohimbinegroup(,2—5)andelectricalstimulat6、ionplusvehiclegroup(一5).Theelectromyogram(EMG)changesofdorsalspinotrapeziusmuscleofCMRinducedbyICofratswereobservedfollowinginterventionwithtreatmentfactorsinvariousgroups.ResultsComparedwithcontrolgroup,EMGcouldbeinhibitedbyelectricalstimulationof10,20and30AinLRN,theinhibitoryeffectwasinc7、reasedwiththeincreasingoftheelectricalstimulationintensityaccompaniedornotaccompaniedwithchangesofbloodpressure;theEMGreducedtO51.41,21.11and1.43ofthecontro1(P<0.001);Comparedwithelectricalstimulation(10“A)group,intrathecalinjectionofadrenergica2receptorantago
4、mentofPhysiology,ShaanxiUniversityofChineseMedicine,Xianyang712046,China;2.DepartmentofPhysiologyandPathophysiology,SchoolofMedicine,Xi’anJiaotongUniversity,Xi’an710061,China)Abstract:ObjectiveTostudythedescendinginhibitoryeffectofthelateralreticularnucleus(LRN)treatedwithelectrica1stimula
5、tionontheeardiosomaticmotorreflex(CMR)inducedbyintrapericardia1capsaicin(IC)inratsanditsmechanism.Methods22maleSDratswererandomlydividedintoelectrica1stimulationgroup(一7),electricalstimulationplus3g·Lyohimbinegroup(n一5),electricalstimulationplus5g·Lyohimbinegroup(,2—5)andelectricalstimulat
6、ionplusvehiclegroup(一5).Theelectromyogram(EMG)changesofdorsalspinotrapeziusmuscleofCMRinducedbyICofratswereobservedfollowinginterventionwithtreatmentfactorsinvariousgroups.ResultsComparedwithcontrolgroup,EMGcouldbeinhibitedbyelectricalstimulationof10,20and30AinLRN,theinhibitoryeffectwasinc
7、reasedwiththeincreasingoftheelectricalstimulationintensityaccompaniedornotaccompaniedwithchangesofbloodpressure;theEMGreducedtO51.41,21.11and1.43ofthecontro1(P<0.001);Comparedwithelectricalstimulation(10“A)group,intrathecalinjectionofadrenergica2receptorantago
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