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ID:41364409
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时间:2019-08-23
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1、VISIONIIS.BuckleyRN,MSNCopyright2008COMMONCAUSESOFVISIONLOSSGlaucomaCataractsDiabeticRetinopathyMacularDegenerationGLAUCOMAGlaucoma:IncludesAGroupOfOcularDisordersCharacterizedByIncreasedIntraocularPressure,OpticNerveAtrophy,AndVisualFieldLoss.IndividualPatientResponseCLASSIFICATIONSPrimaryGlau
2、comaSecondaryGlaucomaAcute&ChronicOpen(Wide)&Closed(Narrow)ETIOLOGYPrimaryOpenAngleGlaucomaOccursAsAResultOfAging.TheDrainageAngleOfTheEyeBecomesLessEfficientWithTime.ThereIsADegenerativeChangeInTheTrabecularMeshwork,ResultingInDecreasedOutflowOfAqueousHumor.90%OfCasesPrimaryAngleClosureGlaucoma
3、IsCausedByPupillaryBlock.TheLensBlocksThePupillaryOpening,PreventingTheFlowOfAqueousHumorIntoTheAnteriorChamberAndDrainingThroughTheTrabecularMeshwork.10%OfTheCases.EYEEXAMEyeExamsMustIncludeTonometryAndAssessmentOfTheOpticNerveHead.NormalPressureis10-21mmhg.AbnormalPressureis22-32mmhg.SECONDARY
4、GLAUCOMASecondaryGlaucomaMayOccurAsAResultOfTraumaInWhichYouGetLensDisplacement,HemorrhageIntoTheAnteriorChamber,Lacerations,AndContusionsWhichCanDisruptTheFlowPatternOfAqueousHumor.PATHOPHYSIOLOGYIntraocularPressureIsDeterminedBy:IntraocularPressureVariesWith:IncreasedPressureInhibitsBloodSuppl
5、yToTheOpticNerveAndRetina.IschemiaAndLossOfFunctionFollow.DIAGNOSTICFINDINGSIncreasedIntraocularPressure.CuppingOrIndentationOfTheOpticNerveHeadOnExam.VisualFieldDefectsUsuallyNotNoticedUntilPeripheralVisionIsSeverelyCompromised.ACUTEANGLECLOSUREGLAUCOMAInAcuteAngleClosureGlaucomaTheAqueousFlow
6、IsObstructedAndIntraocularPressureElevatesMarkedly.Signs&Symptoms:SeverePain,BlurringOrLossOfVision,RainBowHalos,Nausea/VomitingAndCornealEdema.EmergencyRequiringImmediateTreatment.MEDICALMANAGEMENTOFGLAUCOMAEyeMedications:IncreaseOutflowOfOrSuppressTheSecretionOfAqueousHumor.EyeDropRegimenAndD
7、eliveryMethod:SterileTechniqueConsistentTiming3-5MinutesBetweenEachDrop(SameMedication).5-10MinutesBetweenDrops(DifferentMedications).ASSESSMENTDemographicDataMedicationListAllergiesChangesInVisionCopingWithVisionChanges/Los
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