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ID:41144043
大小:917.76 KB
页数:6页
时间:2019-08-17
《植物激素信号通路》由会员上传分享,免费在线阅读,更多相关内容在行业资料-天天文库。
1、一脱落酸ThesimplemodelofABAsignalinstomatalguardcells.Neteffectistheloseofpotassiumionandtheanion(Cl-&soon)incells.cADPR----CyclicADPRibose.IP3----inositol(1,4,5)-trisphosphat.NO----nitricoxide.PA----AcidePhosphatidique.PLC----PhospholipaseC.PLD----Phospholipase
2、D.R----receptor.ROS----reactiveoxygenspecies.S1P----Sphingosine1-phosphate.①BidingofABAandreceptor(inordertoseebetter,onlyshowtheextracellularreceptor);②AfterABAcombiningreceptor,theROSisinducedtoproduce.ThentheCa+channelsisactivatedontheplasmamembrane,ROSi
3、sproducedbyPIDwhichmediatedphosphatidicacid;③TheCa+internalflowcausedinstantaneouschangesofintracellularcalcium.Thatpromotedthereleaseofcalciumfromvacuoles.④ABAistostimulatetheproductofNO,andNOaddcADPR.⑤ABAaddIP3byincludingS1p,theheterogeneousco-polymerandPLD
4、,PLC.⑥TheriseofcADPRandIP3activateothercalciumchannelofthevacuolemembrane,moreCA+releasefromvacuoles.⑦TheriseofintracellularcalciumblockingthechannelsofK+ontheplasmamembrane.⑧TheriseofintracellularcalciumtopromoteopenCl-channelsontheplasmamembrane⑨Protonpumpo
5、ntheplasmamembraneisrestrainedbyABAinducedofintracellularcalciumincreaseandinhibitionofintracellularpHincreased,plasmamembranedepolarizationfurther.⑩K+andanionfromacrosstheplasmamembranevacuolefirstreleasedintothecytoplasm.二生长素AuxinandTIR1/ABFreceptoractivati
6、onafterauxinresponsemodelofgenetranscription。1.Intheabsenceofauxin,AUX/IAAdeterthattheARFtranscriptionactivationcombinedwiththeformationofinactiveformtosuppresstheauxininductionofgenetranscription.AuxinandSCFTIR1unitedthatpromotetheminteractionwithAUX/IAA.2.A
7、uxinactivatedSCFTIR1complexwholemadetheubiquitinmoleculeslinkedtoAUX/IAA.PromotingAUX/IAAdegradebyprotease.AUX/IAAmakedtheARFtranscriptionalactivationproteinremovalanddegradation.3.Inmostoftheinducedbyauxingenes,twoARFproteinmoleculesactivineddimer,tofurtherp
8、romotedgenetranscription.三赤霉素1.GAIcombinedwithaleuronecellsonthesurfaceofthemembranereceptor2.GAreceptorsonthesurfacemadeproteininteractionswiththecellcomplexandheterogeneoustrimerG.3.Wha
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