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1、DeBackerCriticalCare2013,17:247http://ccforum.com/content/17/6/247REVIEWYearinreview2012:CriticalCare-cardiologyDanielDeBacker(3.7±0.6g/dL).Thefluidsdifferedintheirvolumeeffects,Abstractviscosity,andextravasationrateandthesefactorsmayhaveInthisreviewIdiscusskeyresearchpape
2、rsininfluencedmicrocirculatoryperfusionandhencecriticalcardiologyandintensivecarepublishedinCriticalCarehemoglobin.during2012withrelatedstudiespublishedinotherjournalsquotedwheneverappropriate.Thesestudiesaregroupedintothefollowingcategories:Mechanicaltherapiescardiovascul
3、artherapies,mechanicaltherapies,Averyimportanttrialchallengedtheuseofintra-aorticpathophysiologicmechanisms,hemodynamicballoonpump(IABP)counterpulsation[3].Inthisran-monitoring,ultrasoundinrespiratoryfailure,domizedtrialincluding600patientswithcardiogenicmicrocirculation,a
4、ndmiscellaneous.shockcomplicatingacutemyocardialinfarction,IABPfailedtoimprovemortalityat30days(40%versus41%,P=0.69).TherewerenosignificantdifferencesintheReviewtimetohemodynamicstabilization,includingbloodlac-Cardiovasculartherapiestatelevels,andthiswasnotaffectedbythedos
5、eandTheimpactofextracellularacidosisonmyocardialper-durationofcatecholaminetherapy.Surprisingly,therateformanceandresponsivenesstoinotropicstimulationisofcomplicationsdidnotdiffereither,includingbleedingstillthetopicofintensedebate.Whilemildacidosisandischemicevents.Itshou
6、ldbehighlightedthatthistrialseemsnottobeassociatedwithmyocardialdepressionincludedpatientsforwhomanearlyrevascularizationorhyporesponsivenessinisolatedheartsobtainedfromstrategywasplanned,whichmayexplain,atleastinpart,healthyanimals,failingheartsmaybemoresensitivetotherela
7、tivelylowmortalityinbothgroups.Theseresultsacidosis.Isolatedisometricallytwitchinghumanmyocar-maynotapplyinothertypesofcardiogenicshock.dialtrabeculaerecoveredfromend-stagefailingheartsAcasecontrolstudyevaluatedthepotentialroleofde-wereexposedtopH7.40and7.20[1].Thismildmet
8、a-scendingaorticcalcificationsoncomplicationsafterusebolicacidosiscauseda26%depressionint
