资源描述：
《DNP和ATP对Phomopsis longanae Chi侵染的龙眼果实病害发生、能荷状态和呼吸代谢的调控》由会员上传分享，免费在线阅读，更多相关内容在行业资料-天天文库。
1、现代食品科技ModernFoodScienceandTechnology2015,Vol.31,No.5DNP和ATP对PhomopsislonganaeChi侵染的龙眼果实病害发生、能荷状态和呼吸代谢的调控1，21，321，31，31，3陈梦茵，林河通，洪延康，张珅，林艺芬，陈艺晖（1.福建农林大学食品科学学院，福建福州350002）（2.美国佐治亚大学食品科学与技术系，美国佐治亚州格里芬市30223）（3.福建农林大学农产品产后技术研究所，福建福州350002）摘要：与拟茎点霉接种果实相比，DNP能提高拟茎点霉接种果实的病害指数和果皮褐变指数，提高果实呼吸速率和果
2、皮COX、AAO、PPO活性及NAD和NADH含量，降低果皮NADK活性和NADP、NADPH含量及能荷值。而ATP则能降低拟茎点霉接种果实的病害指数和果皮褐变指数，降低果实呼吸速率和果皮COX、AAO、PPO活性及NAD和NADH含量，提高果皮NADK活性和NADP、NADPH含量，维持较高的果皮能荷值。据此认为，DNP处理促进拟茎点霉侵染所致龙眼果实采后病害发生，与DNP加剧能量亏缺、削弱磷酸戊糖呼吸代谢途径及增强COX、AAO和PPO等呼吸末端氧化酶活性有关；而ATP处理延缓拟茎点霉侵染所致龙眼果实采后病害发生，与ATP维持较高的能荷值、增强磷酸戊糖呼吸代谢途径
3、及降低COX、AAO、PPO等呼吸末端氧化酶活性有关。关键词：龙眼；采后病害；拟茎点霉；能荷状态；呼吸代谢文章篇号：1673-9078(2015)5-49-58DOI:10.13982/j.mfst.1673-9078.2015.5.009Regulationof2,4-dinitrophenolandAdenosineTriphosphateonDiseaseDevelopment,EnergyStatus,andRespiratoryMetabolismofPhomopsislonganaeChi-infectedLonganFruit1,21,321,31,31
4、,3CHENMeng-yin,LINHe-tong,HUNGYen-Con,ZHANGShen,LINYi-fen,CHENYi-hui(1.CollegeofFoodScience,FujianAgricultureandForestryUniversity,Fuzhou350002,China)(2.DepartmentofFoodScienceandTechnology,UniversityofGeorgia,1109ExperimentStreet,Griffin,GA30223,USA)(3.InstituteofPostharvestTechnologyo
5、fAgriculturalProducts,FujianAgricultureandForestryUniversity,Fuzhou350002,China)Abstract:TheDNPtreatmentincreasedthecontentsofnicotinamideadeninedinucleotide(NAD)andreducedNAD(NADH),decreasedNADkinase(NADK)activityinthepericarp,andledtolowercontentsofNADphosphate(NADP)andreducedNADP(NAD
6、PH)andalowerlevelofenergycharge.Bycontrast,ATPtreatmentreducedthediseaseindexandpericarp-browningindexforP.longanae-inoculatedlongans,anddecreasedtherespirationrateofthefruitaswellastheactivitiesofCOX,AAO,andPPOinthepericarp.Additionally,ATPtreatmentreducedthecontentsofNADandNADH,increa
7、sedNADKactivityinthepericarp,andledtohighercontentsofNADPandNADPHandahigherlevelofenergycharge.TheseresultssuggestthatDNPtreatmentacceleratesthedevelopmentofpostharvestdiseaseinP.longanae-infectedlongansthroughexacerbatingenergydeficiency,decreasingpentosephosphatepathwayactivi
