山茱萸总苷与多糖对实验性心肌梗死大鼠线粒体保护作用与机制探析

山茱萸总苷与多糖对实验性心肌梗死大鼠线粒体保护作用与机制探析

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时间:2019-02-28

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1、中山大学硕士学位论文山茱萸总苷及多糖对实验性心肌梗死大鼠线粒体保护作用及机制探析ThemechanismofmitochondrialprotectionofCornusofficinalisSieb.etZucconExperimentalAcuteMyocardialInfarctioninRatsMaior:professionalintegratedtraditionalchinesemedicineandwesteInmedicineMastercandidate:chendanSupervisor:ProfessorL

2、iJianjallABSTRACTBackgroundAcutemyocardialinfarctionreferstoasuddencompletecoronaryocclusion,myocardialischemiaocc碱damageandnecrosis,appearingto戳黼chestpain,dynamicECGandmyocardiale11zylncchangesintheclinicalfeaturesofallacuteischemicheartdisease,anditisaclinicallyseve

3、redisease.Westernmedicineandinterventionaltherapyofresearchonitismuchalot,andnumberoftraditionalChinesemedicmeinthetreatmentofstableanginarolehasbeenaf衙med,butresearchesonthemedicmeinthepreventionofacutemyocardialinfarctionandtheprotectiveeffectandmechanismarc传w.Chine

4、semedicmebelievesthatacutemyocardialinfarctionwithinthevirtualinthefight∞usnessarcbasedontheresultofworkandrestproperly,unbridledfoodGaoliangAtsumi,ortheimpassionedinternalinjuriesandotherpathogenic伍ctors.ThedeficiencyofKidneyyinandkidneyyangisthekey.RecentstudiesweTc

5、focusedontraditionalChinesemedicmewhichcanreleasepains,butthat啪Bushengubiaowasless.Comuscanstrongfiverandkidney,anditiscriticalinther{杈2uecoronaryhemdiseaseanditshowsa3up.oreffect.Andacutemyocardialischerniamitochondrialenergydepletion,releaseofreactiveV中山大学硕士学位论文山茱萸总

6、苷及多糖对实验性心肌梗死大鼠线粒体保护作用及机制探析oxygenspecies,induceapoptosisandinjury,andtheyareimportantpathologicalchanges.ThereforeWOconcludethatthereisdelicatelinkbetwcellmitoehondriaandkidney.Throughresearchollbeforeandafteracutemyocardialinfarctionandactiveingredientsmitoehondrialst

7、ructureandfunctionofthenumberofchangesoccurredbeforeandaltercOrllUStreating,wehopetorevealthenatureofstrengthenkindey.Purposeofthisstudyistoexplaintheessenceofstrengtheningkidney.ThroughresearchesOilthecorlluswhichCAlllstrengthenliverandkidneyplayedapartOnmyocardialpr

8、otectionOilrats,andthechangesofnumberofpost-acutemyocardialinfarctioncardiacmitoehondrial,structureandfunction,andwhethertha

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