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1、病因VascularMalformationsSimilarly,cavernomasarerarelydescribedasacauseofSAH.However,superficialandleptomeningealcavernomasmaybethesourceofcSAH.RCVSRCVSVasculitidesVasculitidesInfectiousOriginCorticalSAHmaybeseenafterruptureofinfectiousaneurysms(causedbyinfec
2、tiveendocarditis,meningitis,orrarelycrypto-genically)(Fig5).Headacheisdiffuseandvague,ratherthanthetypicalexcruciating极痛苦headacheofnoninfectiousaneurysmalSAH.Usually,therearealsogeneralsymptoms(eg,anorexia,weightloss,malaise,orfever)Ininfectiveendocarditis,
3、MRimagingmightshowassociatedlesions(ischemic,microbleeds,brainabscesses).DSAmightbeusedforthediagnosisofdistalarteriallesionsandfortherapeuticpurposes.InfectiousOriginInfectiousOriginInfectiousOriginMoyamoyaAccordingtoarecentclassification,patientswithwellr
4、ecognizedassociatedconditions(eg,sicklecelldisease，neurofibromatosistype1,cranialtherapeuticirradiation,andDownsyndrome)areconsideredashavingaMoyamoyasyndrome,whilepatientswithnoknownassociatedriskfactorsaresaidtohaveMoyamoyadiseaseMoyamoyaHigh-GradeStenosi
5、sFig6.Pialvasodilation.A,AxialGRET2imageshowsaleftfrontalsulcalSAH(blackarrowhead),possiblylocatedinthe“watershed”territorybetweentheanteriorandthemiddlecerebralarteries.B,Axialmaximum-intensity-projectionreconstructionofCTAshowsanasymmetryofthedistalarteri
6、es,infavorofleftpialvasodilation.C,Frontalprojectionof3DangiographyoftheleftcarotidarteryrevealsaseverestenosisattheoriginoftheM2branch(whitearrowhead).Fig.1ACTdemonstratesaleftinferiortemporalSAH.BThisisconfirmedonMRI.CSeveralareasofrestricteddiffusionares
7、eenintheleftMCAregion.DAngiographydemonstratestightbilateralinternalcarotidstenosesposteriorreversibleencephalopathysyndromecerebralamyloidangiopathycerebralamyloidangiopathyA,AxialbrainCTscanshowsasubtleleftrolandichyperattenuationfavoringminimalSAH(whitea
8、rrowhead).BandC,AxialGRET2imagesshowlefttemporallobarhemorrhage,multiplemicrobleeds,andcorticalhemosiderosis.D,AxialFLAIRimageobtainedafter7monthsshowsanewasymptomaticSAHinaleftparietalsulcus(whitearro
