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1、英文摘要ThecorrelationbetweenVasopressin，Urotensin—IIandHyponatremiainlivercirrhosiswithascitesABsTRACTHyponatremiaiswhensenlmsodiumiSlessthanorequaltO135mmol／L．HyponatrerIliaiscommonintheascitesoflivercirrhosis．CombiningthedomesticreportsofChina,theincidencerateoflivercirrhosisasc
2、itescomplicatedhyponatremiais50--60％，30％byaseriesofforeignreports．Thehyponatremiaoflivercirrhosisasciteshasnospecificclinicalmanifestation，anditssymptomshavesomethingtodowithcomplication,severityofhyponatremia，age，sex，ete．Hyponatremiahastwocases，withoutsymptomsormanifestsitself
3、,forexamplenausea,debility,disorientation,headache，lethargy,blurredvision,evenencephalopathyincludingmindconfusing，delire，epilepticattack,ere．Peopleoftenmisinterpretthese器hepaticcoma．Whenseu衄sodiumislessthanorequalto15mmol／L，itsclinicalmanifestations(debility,headache，musclecon
4、vulsion,apthy,lethargy,mentalsubnormality,delirium，convulsion，coma)areclear,andeven10wnatriumencephalopathyleadtocardiorespiratoryarrest．Whenlivercirrhosiscomplicatedacutelownatriumsyndrome(ALNS)，itsclinicalmanifestationsareoftennausea,vomiting,debility,ere．Ands@veresubjectshav
5、esymptomsofunorientation，blurredvision，coma,andperipheralcirculatoryfailure．Whensertlnl荚文摘要sodiumisIesst11a11orequaltO135mm01／Landosmoticpressureofplasmaismorethan280mmol／L，itmayberegardedaspseudo—hyponatremia．ThereflsonflmayberelatedtOhyperglycemia，hyperlipemia，etc．Thegradingo
6、fhyponatremiaisthelighthyponatremiawhenserumsodiumis130～135mmol／L，themiddlehyponatremiawhenserBinsodium’is125-130mmoFLandtheseverehyponatremiawhenserumsodiumislessthanorequalto125mmol／L．Vasopressin(VP)canregulatetheconcentrationofufineinmedullsintemaofkidney,andincreasetheperme
7、abilitytowaterincollectingduct．Inlivercirrhosis，thesecretionofVPisstimulatedbynon-osmolarity,forthechangeofhemorrheology,includingthedecreaseofcentralhypovolemiaandtheincreaseofperipheralvascularresistanceandthedecreaseofeffectivebloodvolumeleadstOnon-osmolaritysecretionofVP．Al
8、thoughplasmaosmoticpressuredecreased，stimulationofnono
