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ID:59716656
大小:1.75 MB
页数:18页
时间:2020-11-20
《生物医学U3B资料.ppt》由会员上传分享,免费在线阅读,更多相关内容在教育资源-天天文库。
1、生物医学U3BPara.1:TheeffectsofinflammationInflammation:thereactionofvascularizedtissuetolocalinjury.effectsofinflammation:UndesirableyetbeneficialPara.2:Thecausesofinflammationthecausesofinflammation:infectiousmicroorganisms,trauma,surgery,causticchemicals,extremesofheatandcold,ischemicdamagetotissue
2、sPara.3:Similarresponses“…initiatedbyavarietyofinjuriousagents...”initiate:cause/startsthE.g.Cardiovasculardiseasescanbeinitiatedbyafattydiet.油腻的饮食会引发心血管疾病。injuriousagents:损伤因子Para.4:Namingofinflammation-itisusedtodenoteaninflammationExamplesListofinflammationsBodypartBodypartAppendicitisAppendix
3、ArthritisJointBronchitisBronchiGastritisStomachNephritisKidneyPneumonitis/PneumoniaLungsHepatitisLiverPericarditisPericardiumneuritisnerveExamplesSigns:Rubor/rednessTumor/swellingCalor/heatDolor/painFunctiolaesa/lossoffunctionPara.5:Descriptionofacuteinflammationcardinalsignsofinflammation:发炎主征ag
4、eneralalarmandrecruitmentsystem:全身的报警和召集系统acute-phaseresponse:急性期反应plasmaproteins:血浆蛋白Para.6:ManifestationofacuteinflammationVascularresponseCellularbloodresponseThingsincommon:releaseofchemicalmediatorsinflammatoryexudatesThehemodynamicandwhitebloodcellresponsescontributetotheinflammatoryexudate
5、s,theextravascularinfluxoffluidcontaininghighconcentrationsofproteins,salts,cells,andcellulardebris.influx:thearrivaloflargenumbersofpeopleorlargeamountsofmoney,goodsetc,especiallysuddenly(>influxof)大量涌入血液动力和白细胞反应促使炎症渗出物的形成。它是从血管渗出流到炎症部位的体液,含有高浓度的蛋白质、盐、细胞、以及细胞碎片。Paras.7-9:vascularresponseHowdoest
6、hevascularresponseoccur?Whycanvascularresponsebenefitthehost?Whatarethethreetypesofvascularresponse?Process:Momentaryconstriction→vasodilation→congestion→redness/warmness→increaseincapillarypermeability→swelling→pain/impairedfunctionBenefit:DilutelocalizethreepatternsofvascularresponsesImmediatet
7、ransientImmediatesustainedDelayedPara.10:cellularresponseFeature:MovementofleukocytesintotheareaofinjuryTheselonger-livedphagocyteshelptodestroytheagent,aidsignalingofspecificimmunity,andservetoresolvetheinflammatorypr
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